BCIA Continuing Education Posts

FOXP1 and FOXP2 mutations have been implicated in neurodevelopmental disorders and autism spectrum disorder (ASD).

The 1996 Task Force Report remains deeply influential today.

Spindling excessive beta (SEB) represents a critical neurophysiological marker of cortical dysregulation identifiable on the EEG.

Breathing ensures healthy CO2 levels. The main functions of breathing are gas exchange and acid-base (pH) regulation.

Future standards must include open algorithms and population-level validation to increase clinical trust.

AI in mental health care can increase accessibility and offer scalable support, particularly through chatbots and virtual therapists.

The SCAN integrates cognition and movement.

I wrote this post to clear up confusion about frequency and amplitude, and give you a basic format for thinking about the EEG signal.

The IQCB guidelines formalize data collection, inspection, and analysis procedures to improve qEEG reliability and clinical acceptance.

The thalamus may act as a neural gatekeeper, filtering stimuli that enter conscious awareness.

Prospective value coding reconciles previous models of serotonin function.

Schizophrenia represents multiple disorders—"schizophrenias"—each potentially with distinct biological underpinnings.

The temporal and parietal lobes are the earliest affected, and the frontal and occipital lobes are the latest.

ADHD and ODD are distinct disorders with unique causes, symptoms, and treatment needs.

ADHD is associated with hypocoherence in major neural networks, particularly the DMN, SalN, and AttDN.

SNS activity is too fast and irregular to be a primary driver of VLF rhythms.

Language allows us to share our thoughts and ideas with others, rather than being essential for thinking.

Integrating biofeedback and neurofeedback represents a promising frontier in physiological self-regulation approaches.

The medical community has mistakenly viewed transient ischemic attacks as relatively harmless events.

Environmental and lifestyle factors accounted for 17% of disease mortality risk, while genetics contributed just 2%.