BCIA Continuing Education Posts

A global drop in amplitude suggests either a disruption in metabolic support—such as mitochondrial failure—or widespread cortical inhibition.

This study makes a valuable contribution to our understanding of the effects of neurofeedback on ADHD-related brain activity.

Autism is not one condition but likely represents "autisms," different biological pathways leading to outwardly similar traits.

At its core, ADHD is not about lacking attention—it's about struggling to control it.

Stimulant treatment does not reliably translate into better grades or sustained academic gains.

EEG biomarkers offer incremental validity over behavioral observation alone.

Spindling excessive beta (SEB) represents a critical neurophysiological marker of cortical dysregulation identifiable on the EEG.

I wrote this post to clear up confusion about frequency and amplitude, and give you a basic format for thinking about the EEG signal.

ADHD and ODD are distinct disorders with unique causes, symptoms, and treatment needs.

ADHD is associated with hypocoherence in major neural networks, particularly the DMN, SalN, and AttDN.

Integrating biofeedback and neurofeedback represents a promising frontier in physiological self-regulation approaches.

Fetal exposure to acetaminophen may increase the likelihood of developing attention-deficit/hyperactivity disorder (ADHD).

The study found a significant reduction in life expectancy for adults with ADHD.

Consider hormonal cycles when diagnosing and treating ADHD in women.